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Such episode is widely believed to be a self-limiting condition, which does not necessitate prolonged follow-up or treatment.However, a recent study, examining the long-term outcome in rotator cuff calcific tendonitis, demonstrated that after the mean follow-up of 14 years, about 55% of 194 subjects had impaired shoulder function, with more than 40% severely impaired .
In most patients with crystal-induced enthesopathy/tendinopathy, entheseal or tendineal calcifications can be easily seen on X-ray films or ultrasonographic examination.
Diagnosed with acute calcific tendonitis, these patients usually receive short-term treatment with nonsteroidal anti-inflammatory drugs, glucocorticoid injection, and/or a course of physical therapy.
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Recent data cast a new light on the role of enthesopathy in the large body of rheumatic disorders.
Entheses are sites of attachment of tendons, ligaments, fasciae, and articular capsules to bones.
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With the aging of the population and an increased prevalence of metabolic syndrome, both serving as a backdrop to crystal-associated enthesopathy, we see this quantitatively as a major rheumatologic condition to be dealt with.
Yet, paradoxically, the majority of these patients are treated by orthopedic surgeons.